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"Latar Belakang: Salah satu faktor yang menentukan keberhasilan restorasi proksimal resin komposit adalah adaptasi yang rapat tepi restorasi dan dinding gingiva kavitas. Restorasi resin komposit akan mengalami kontraksi saat polimerisasi sehingga terdapat celah antara tepi restorasi dan kavitas. Celah ini dapat menimbulkan kebocoran mikro sehingga menyebabkan bakteri, cairan, molekul, dan ion masuk kedalamnya. Penelitian ini bertujuan untuk menganalisis perbedaan tingkat kebocoran mikro dinding gingiva restorasi proksimal resin komposit nanohibrid antara teknik inkremental, bulk-fill yang diaktivasi sonik, dan tanpa aktivasi sonik.Metode: Kavitas kelas II dipreparasi pada tiga puluh gigi premolar rahang atas dan bawah, kemudian dibagi menjadi tiga kelompok. Kelompok pertama ditumpat dengan RK bulk-fill yang diaktivasi sonik, kelompok kedua dengan RK bulk-fill tanpa aktivasi sonik, dan kelompok ketiga dengan RK yang diletakkan secara inkremental. Selanjutnya spesimen direndam dalam air distilasi selama 24 jam dan kemudian dilakukan uji thermocycling, yang diikuti perendaman dalam metilen biru 1% selama 24 jam. Gigi selanjutnya dibelah longitudinal dan dilakukan pengamatan menggunakan mikroskop stereo pembesaran 25x dan dinilai dalam skala ordinal (0-3). Analisis statistik dilakukan dengan uji Kolmogorov-Smirnov.Hasil: Tidak terdapat perbedaan bermakna secara statistik di antara tiga kelompok.Kesimpulan: Tidak ada satupun dari kelompok RK bulk-fill yang diaktivasi sonik, bulk-fill tanpa aktivasi sonik dan yang diletakkan secara inkremental yang dapat menghilangkan kebocoran mikro pada dinding gingiva kavitas kelas II.Kata kunci: kebocoran mikro, teknik peletakan resin komposit, kontraksi

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Background One of the factor that determine the success of proximal composite restoration is a good marginal adaptation at the interface area Composite resin will undergo contraction during polimerization which may result gap formation between the wall cavity and restoration. The gap can cause a microleakage and resulting a passage for bacteria fluid molecules and ions The purpose of this study is to analize the microleakage of gingival wall nanohybrid composite restoration that filled using sonic activated bulk bulk without sonic activation and incremental techniques.

Methods Standardized class II cavities were prepared on 30 extracted human upper and lower human teeth and randomly assigned to three groups The first group were filled with sonicactivated bulk fill composite resin the second group were filled with bulk fill composite resin without sonic activation and the third group were filled incrementally. The specimens were stored in distilled water for 24 hours and then subjected to thermocycling followed by immersion in 1 methylene blue dye for 24 hours. The teeth were sectioned longitudinally and evaluated for microleakage under 25x magnification stereomicroscope and scored in ordinal scale 0 3 Statistical analysis was performed with the Kolmogorov Smirnov test.

Results There was no statistically significant difference among the three groups Conclusion None of the the techniques was capable of eliminating the microleakage on gingival wall cavity preparations."

"ABSTRAK Latar Belakang. P.gingivalis merupakan bakteri periodonsium yang dapatberpindah ke saluran akar pada lesi endoperio kombinasi murni. Jumlah danproporsinya berperan terhadap keparahan penyakit karena sifat virulensinya.Tujuan: Menganalisis jumlah dan proporsi P.gingivalis saluran akar dan poketlesi endoperio kombinasi murni, lesi endo primer, dan lesi perio primer. Metode:16 sampel diambil dari 3 kelompok, kuantifikasi dengan PCR real-time. Hasil:Jumlah P.gingivalis terbesar pada poket lesi perio primer, proporsi P.gingivalisterbesar pada saluran akar lesi endo primer. Jumlah dan proporsi P.gingivalistidak berbeda bermakna antara saluran akar dan poket lesi endoperio kombinasimurni. Kesimpulan. Jumlah dan proporsi P.gingivalis sebanding antara saluran akar dan poket lesi endoperio kombinasi murni.ABSTRACT Background : P.gingivalis is a periodontium pathogen that can be transferred tothe root canal of the true combined endoperio lesions. Quantity and proportiontakes part in the severity of the disease because of its virulence. Objective: toanalyze the quantity and proportion of P.gingivalis in the root canals and pocketof true combined endoperio lesions, primary endodontic lesions, and primaryperiodontal lesions. Methods 16 samples were taken from 3 groups,quantification with real-time PCR. Results : P.gingivalis largest quantity wasfound in pockets of primary periodontal lesions, P.gingivalis largest proportionwas found in root canals of primary endodontic lesions. There were no significantdifferences in quantity and proportion of P.gingivalis in root canals and pockets oftrue combined endoperio lesions. Conclusion. Quantity and proportion ofP.gingivalis were comparable between the root canals and pockets of true combined endoperio lesions.;Background : P.gingivalis is a periodontium pathogen that can be transferred tothe root canal of the true combined endoperio lesions. Quantity and proportiontakes part in the severity of the disease because of its virulence. Objective: toanalyze the quantity and proportion of P.gingivalis in the root canals and pocketof true combined endoperio lesions, primary endodontic lesions, and primaryperiodontal lesions. Methods 16 samples were taken from 3 groups,quantification with real-time PCR. Results : P.gingivalis largest quantity wasfound in pockets of primary periodontal lesions, P.gingivalis largest proportionwas found in root canals of primary endodontic lesions. There were no significantdifferences in quantity and proportion of P.gingivalis in root canals and pockets oftrue combined endoperio lesions. Conclusion. Quantity and proportion ofP.gingivalis were comparable between the root canals and pockets of true combined endoperio lesions.;Background : P.gingivalis is a periodontium pathogen that can be transferred tothe root canal of the true combined endoperio lesions. Quantity and proportiontakes part in the severity of the disease because of its virulence. Objective: toanalyze the quantity and proportion of P.gingivalis in the root canals and pocketof true combined endoperio lesions, primary endodontic lesions, and primaryperiodontal lesions. Methods 16 samples were taken from 3 groups,quantification with real-time PCR. Results : P.gingivalis largest quantity wasfound in pockets of primary periodontal lesions, P.gingivalis largest proportionwas found in root canals of primary endodontic lesions. There were no significantdifferences in quantity and proportion of P.gingivalis in root canals and pockets oftrue combined endoperio lesions. Conclusion. Quantity and proportion ofP.gingivalis were comparable between the root canals and pockets of true combined endoperio lesions.;Background : P.gingivalis is a periodontium pathogen that can be transferred tothe root canal of the true combined endoperio lesions. Quantity and proportiontakes part in the severity of the disease because of its virulence. Objective: toanalyze the quantity and proportion of P.gingivalis in the root canals and pocketof true combined endoperio lesions, primary endodontic lesions, and primaryperiodontal lesions. Methods 16 samples were taken from 3 groups,quantification with real-time PCR. Results : P.gingivalis largest quantity wasfound in pockets of primary periodontal lesions, P.gingivalis largest proportionwas found in root canals of primary endodontic lesions. There were no significantdifferences in quantity and proportion of P.gingivalis in root canals and pockets oftrue combined endoperio lesions. Conclusion. Quantity and proportion ofP.gingivalis were comparable between the root canals and pockets of true combined endoperio lesions.;Background : P.gingivalis is a periodontium pathogen that can be transferred tothe root canal of the true combined endoperio lesions. Quantity and proportiontakes part in the severity of the disease because of its virulence. Objective: toanalyze the quantity and proportion of P.gingivalis in the root canals and pocketof true combined endoperio lesions, primary endodontic lesions, and primaryperiodontal lesions. Methods 16 samples were taken from 3 groups,quantification with real-time PCR. Results : P.gingivalis largest quantity wasfound in pockets of primary periodontal lesions, P.gingivalis largest proportionwas found in root canals of primary endodontic lesions. There were no significantdifferences in quantity and proportion of P.gingivalis in root canals and pockets oftrue combined endoperio lesions. Conclusion. Quantity and proportion ofP.gingivalis were comparable between the root canals and pockets of true combined endoperio lesions."

"Latar belakang: Penderita hipertensi yang rutin mengonsumsi obat antihipertensi memiliki resiko lebih tinggi untuk mengalami karies gigi. Kapasitas buffer menjadi salah satu faktor protektif utama gigi terhadap karies gigi karena kapasitas buffer yang ada di dalam saliva berfungsi sebagai penetralisir pH dalam rongga mulut. Mengonsumsi obat antihipertensi dapat menimbulkan efek samping berupa pengurangan sekresi saliva (serostomia). Kapasitas buffer saliva berhubungan erat dengan laju aliran saliva pada rongga mulut. Ketika laju aliran saliva pada individu menurun, maka kapasitas buffer pada juga akan menurun. Tujuan: Menganalisis perbedaan kapasitas buffer saliva terstimulasi pada kelompok yang mengonsumsi obat antihipertensi amlodipin dan yang tidak mengonsumsi obat antihipertensi amlodipn. Metode: Saliva terstimulasi 30 individu yang mengonsumsi obat antihipertensi amlodipin (subjek) dan 30 individu yang tidak mengonsumsi obat antihipertensi amlodipin (kontrol) dikumpulkan dengan cara mengunyah non-flavoured parafin wax untuk kemudian diukur kapasitas buffer nya dengan menggunakan buffer test foil pack. Hasil: Terjadi perbedaan bermakna pada kapasitas buffer saliva tersimulasi antara kelompok subjek dan kelompok kontrol. Kesimpulan: kapasitas buffer dalam saliva terstimulai pada kelompok subjek [6.0 ( 4 – 9 )] lebih rendah dibandingkan dengan kelompok kontrol [9.5 ( 7 – 12 )] yang mana berdasarkan analisis statistik, hal tersebut berbeda bermakna secara signifikan.

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Background: Hypertension patients who routinely take antihypertensive drugs have a higher risk to suffer from dental caries. Buffer capacity is one of the main protective factors of teeth against dental caries because buffer capacity in saliva functions as a neutralizing pH in the oral cavity. Taking antihypertensive drugs can cause side effects such as reduced salivary secretion (xerostomia). Buffer capacity in saliva is related to the flow rate of saliva in the oral cavity. When the salivary flow rate in an individual decreases, the buffer capacity of that individual will also decrease. Objective: to analyze the difference of buffer capacity stimulated saliva in the group whose taking amlodipine and those who did not take amlodipine. Method: Stimulated saliva of 30 individu who consumed amlodipine (subjects) and 30 individu who doesn’t experience hypertensive (controls) were collected by chewing non-flavored paraffin wax and then buffer capacity was measured by using a buffer test foil pack. Result: There were significant differences in the buffer capacity of stimulated saliva between the subject and control groups. Conlusion: the buffer capacity in the subject group [6.0 (4-9)] is lower than the control group [9.5 (7-12)] which is significantly different by statistical analysis."

"ABSTRAK Latar Belakang : Enterococcus faecalis merupakan bakteri yang mampu membentuk biofilm dan banyak ditemukan pada kasus kelainan periapeks.Tujuan : Melihat daya antibakteri campuran triantibiotik dibandingkan denganklorheksidin 2% terhadap E. faecalis dalam biofilm. Metode : Menilai kekeruhanlarutan E. faecalis dalam biofilm pasca pemaparan bahan uji, dengan ELISAreader. Hasil : Terdapat daya antibakteri campuran triantibiotik terhadap biofilmbakteri E. faecalis tetapi tidak terdapat perbedaan bermakna dengan klorheksidin2% (p>0.05). Kesimpulan : Daya antibakteri campuran triantibiotik terhadapbiofilm E. faecalis sebanding dengan klorheksidin 2%. ABSTRACT Background : Enterococcus faecalis has the ability to form biofilm and is oftenfound in cases of periapical lesions. Aim: To analyze the effectivity of triantibioticmixture compared to 2% chlorhexidine against biofilm of E. faecalis. Method :Score the turbidity of E. faecalis in biofilm after immersion in antibacterialagent, with ELISA reader. Result : Triantibiotic mixture has antibacterialeffectivity against E. faecalis biofilm but has no significant difference compared to2% chlorhexidine (p>0.05). Conclusion : Antibacterial effectivity of triantibioticmixture against E. faecalis biofilm is equal to2% chlorhexidine. ;Background : Enterococcus faecalis has the ability to form biofilm and is oftenfound in cases of periapical lesions. Aim: To analyze the effectivity of triantibioticmixture compared to 2% chlorhexidine against biofilm of E. faecalis. Method :Score the turbidity of E. faecalis in biofilm after immersion in antibacterialagent, with ELISA reader. Result : Triantibiotic mixture has antibacterialeffectivity against E. faecalis biofilm but has no significant difference compared to2% chlorhexidine (p>0.05). Conclusion : Antibacterial effectivity of triantibioticmixture against E. faecalis biofilm is equal to2% chlorhexidine. ;Background : Enterococcus faecalis has the ability to form biofilm and is oftenfound in cases of periapical lesions. Aim: To analyze the effectivity of triantibioticmixture compared to 2% chlorhexidine against biofilm of E. faecalis. Method :Score the turbidity of E. faecalis in biofilm after immersion in antibacterialagent, with ELISA reader. Result : Triantibiotic mixture has antibacterialeffectivity against E. faecalis biofilm but has no significant difference compared to2% chlorhexidine (p>0.05). Conclusion : Antibacterial effectivity of triantibioticmixture against E. faecalis biofilm is equal to2% chlorhexidine. ;Background : Enterococcus faecalis has the ability to form biofilm and is oftenfound in cases of periapical lesions. Aim: To analyze the effectivity of triantibioticmixture compared to 2% chlorhexidine against biofilm of E. faecalis. Method :Score the turbidity of E. faecalis in biofilm after immersion in antibacterialagent, with ELISA reader. Result : Triantibiotic mixture has antibacterialeffectivity against E. faecalis biofilm but has no significant difference compared to2% chlorhexidine (p>0.05). Conclusion : Antibacterial effectivity of triantibioticmixture against E. faecalis biofilm is equal to2% chlorhexidine. "

"Latar Belakang: Affected dentin merupakan lapisan yang masih dapat terjadi remineralisasi karena masih terdapat ikatan silang kolagen dan prosesus odontoblastik yang masih vital yang merupakan syarat terjadinya remineralisasi. Terdapat dua metode remineralisasi, yaitu metode konvensional dan guided tissue remineralization GTR . Pada metode konvensional hanya dapat terjadi remineralisasi secara ekstrafibrillar. Sedangkan pada metode GTR memiliki keunggulan yaitu dapat terjadi remineralisasi secara ekstrafibrillar dan intrafibrillar. Beberapa penelitian melaporkan bahwa mineralisasi intrafibrillar dapat meningkatkan properti mekanis dari dentin.Pada metode GTR dibutuhkan peran protein non kolagen, yaitu DMP 1. Namun karena proses karies, maka sebagian DMP 1 mengalami kerusakan sehingga dibutuhkan material analog protein non-kolagen, salah satunya adalah Carboxymethyl Chitosan/Amorphous Calcium Phosphate CMC/ACP . Tujuan: Mengevaluasi terjadinya remineralisasi intrafibrillar pada permukaan demineralized dentin setelah aplikasi material analog protein non-kolagen CMC/ACP. Metode: Empat kelompok dilakukan demineralisasi buatan, dalam satu gigi terdapat dua kavitas, salah satu kavitas diaplikasikan material CMC/ACP, sedangkan kavitas lainnya tidak diaplikasikan CMC/ACP. Sampel diperiksa pada hari ke-7 dan ke-14 dengan Transmission Electron Microscope TEM . Hasil: Terlihat peningkatan kadar kalsium dan fosfat setelah aplikasi CMC/ACP pada hari ke-7 dan ke-14.Kesimpulan: CMC/ACP memiliki potensi untuk meremineralisasi demineralized dentin.

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Background Affected dentin is a layer which can be remineralized due to the presence of cross linked collagen and a living odontoblastic process a key to remineralization. There are two methods of remineralization convensional and guided tissue remineralization GTR . In conventional methods, only extrafibrillar remineralization occurs. GTR resulting intrafibrillar and extrafibrillar remineralization. Intrafibrillar remineralization improves physical properties of dentin. GTR is a method of collagen dentin remineralization using non collagen protein, Dentin Matrix Protein 1 DMP 1 . DMP 1 is damaged due to caries process. Carboxymethyl Chitosan Amorphous Calcium Phosphate CMC ACP has similar function with DMP 1. Aim To evaluate intrafibrillar remineralization on demineralized dentin after application non collagen protein analog CMC ACP. Method Four groups performed artificial demineralization two of which applied CMC ACP material. Whereas, the other group was not applied CMC ACP. Evaluation of intrafibrillar remineralization with Transmission electron Microscope TEM . Result After 7 days and 14 days CMC ACP application, intrafibrillar remineralization was observed in the gap zone. Conclusion CMC ACP has a potential for intrafibrillar remineralization on demineralized dentin."

"Latar Belakang: Karies gigi merupakan penyakit yang dialami oleh masyarakat Indonesia, disebabkan oleh proses demineralisasi jaringan keras gigi. Saliva adalah faktor perlindungan alami terhadap karies yang dapat distimulasi oleh pengunyahan permen karet yang mengandung xylitol.Tujuan: Mengetahui pengaruh pengunyahan permen karet yang mengandung xylitol terhadap laju aliran saliva.Metode: Penelitian menggunakan metode cross-over. Total subyek 30 anak diberikan 3 macam perlakuan (pengunyahan parafin, 2 buah dan 4 buah permen karet yang mengandung xylitol) selama 5 menit. Pemeriksaan menggunakan gelas ukur salivary test kit GC.Hasil penelitian: Uji statistik ANOVA 1 arah menunjukkan perbedaan bermakna (p < 0,05) antara semua kelompok.Kesimpulan: Terjadi peningkatan laju aliran saliva dengan pengunyahan permen karet yang mengandung xylitol dan peningkatan terjadi seiring dengan penambahan jumlah permen karet yang mengandung xylitol.

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Background: Dental caries is a common oral disease to the Indonesians, which is caused by demineralization of tooth?s hard tissues. Saliva is a natural protective agent against caries that can be stimulated by chewing xylitol chewing gum.Objective: To identify the effect of chewing xylitol chewing gum on salivary flow rate.Method: Cross-over method. Thirty children having decayed and filled tooth ≥ 3 teeth are given 3 kinds of treatment (chewing paraffin, chewing 2 pieces, and chewing 4 pieces of xylitol chewing gum) on a 5-minute basis. Salivary flow rates are evaluated using GC salivary test kit metric cups.Result: Statistical evalution of one-way ANOVA shows significant differences (p<0,05) between all groups.Conclusion: There is an increase of salivary flow rate after chewing xylitol chewing gum, and the increase is proportional to the amount of the chewing gum."

"Latar Belakang : Karies adalah penyakit gigi yang sering terjadi di Indonesia. Saliva berperan dalam terjadinya karies. Saat ini xylitol dapat mencegah karies dan belum ada penelitian yang melihat pengaruh xylitol terhadap pH saliva.Tujuan: mengetahui pengaruh mengunyah permen karet yang mengandung xylitol terhadap perubahan nilai pH saliva. Metode: 30 anak berusia 10-12 tahun diberikan tiga perlakuan: mengunyah parafin, 2 permen karet xylitol, dan 4 permen karet xylitol selama 5 menit. Data dianalisis dengan uji statistik dengan p<0,05.Hasil: Kelompok sebelum dan sesudah parafin, 2 xylitol, serta 4 xylitol didapat masing-masing nilai p=0,000; kelompok sesudah parafin dengan sesudah 2 xylitol (p=0,472); kelompok sesudah parafin dengan sesudah 4 xylitol (p=0,000).Kesimpulan: Peningkatan pH saliva terjadi seiring dengan bertambahnya jumlah permen karet xylitol.

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Background: Dental caries is one of the common dental health problem in Indonesia. Saliva has a role in caries process. Recently, xylitol usage can prevent dental caries and no research has studied the effect on salivary pH.Objective: Identify the effect of xylitol xhewing gum on salivary pH.Method: 30 subjects aged between 10-12 years will get three kinds of treatment (cross-over method): chewing paraffin, 2 pieces of xylitol chewing gum, and 4 pieces of xylitol chewing gum on a 5 minute basis. The research data will be evaluated with statistic analysis (p<0,05).Result: Before and after parafin, 2 xylitol, and 4 xylitol p=0,000; between parafin and 2 xylitol p=0,472; between parafin and 4 xylitol p=0,000.Conclusion: The increase of salivary pH is proportional to the amount of the gum chewed."

"Latar Belakang: Karies gigi merupakan penyakit yang sangat umum terjadi di Indonesia. Faktor - faktor yang menyebabkan karies gigi antara lain peningkatan retensi dan akumulasi plak, asam organic pada gigi, penggunaan flour, frekuensi diet asam dan karbohidrat, serta factor pelindung dari pelikel dan saliva. Salah satu cara untuk mencegah terjadinya karies gigi adalah dengan penggunaan xylitol sebagai pemanis pengganti gula karena tidak bisa difermentasi oleh bakteri.Tujuan: Mengetahui pengaruh mengunyah sejumlah permen karet xylitol terhadap kapasitas buffer saliva. Metode: Menggunakan cross-over, melibatkan 30 anak berusia 10-12 tahun yang memiliki gigi karies atau ditambal ≥ 3. Setiap subyek diberikan tiga perlakuan, yaitu: pengunyahan parafin, pengunyahan 2 buah permen karet xylitol, dan pengunyahan 4 buah permen karet xylitol. Pemeriksaan kapasitas buffer saliva menggunakan Salivary Check merek GC. Data hasil penelitian ini dianalisis dengan menggunakan pengukuran statistic Kruskall- Wallis dan U Mann - Whitney.Hasil: Terdapat perbedaan yang bermaknaan nilai kapasitas buffer setelah pengunyahan antara parafin, 2 buah xylitol, dan 4 buah xylitol (p<0,05). Simpulan: Terjadi peningkatan kapasitas dapar saliva setelah mengkonsumsi permen karet yang mengandung xylitol dan peningkatan ini terjadi seiring dengan bertambahnya jumlah permen karet yang mengandung xylitol.

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Background: Dental caries is a very common disease in Indonesia. Factors that cause dental caries are increased retention and accumulation of plaque, increased production of organic acids at tooth interface, usage of fluoride, frequency carbohydrate and dietary acids, and the protective factors of pellicle and saliva. One of ways to prevent dental caries is by using xylitol as alternative sweeteners because bacteria can?t fementized it.Objectives: To identify the effect of chewing gum that contains xylitol on salivary buffer capacity. Method: Using cross-over method, involving 30 subject aged between 10-12 years who have carious and restored teeth ≥ 3. Every subject will get three kinds of treatment for 5 minutes: chewing paraffin, chewing 2 xylitol chewing gum, and chewing 4 xylitol chewing gum. Salivary buffer capacity is checked with Salivary Test kit from GC. The research data was analized with Kruskall-Wallis and U Mann Whitney.Result : There were reasonable level of significance of salivary buffer capacity after chewing paraffin, 2 pieces of xylitol, and 4 pieces of xylitol (p<0,05).Conclusion: Salivary buffer capacity increased after consuming chewing gum that contains xylitol and the increasing along with the chewing gum amount."

"Ketahanan ikatan resin komposit-dentin merupakan salah satu penentu keberhasilan restorasi resin komposit. Tujuan penelitian ini adalah menganalisis efek klorheksidin terhadap degradasi kekuatan ikat resin kompositdentin. Metode: Dua puluh empat sampel dentin yang diambil dari mahkota gigi premolar, dibagi menjadi tiga kelompok yang diberikan perlakuan berbeda. Kelompok I diberi perlakuan bahan bonding tanpa klorheksidin, kelompok II diberi perlakuan klorheksidin dan bonding, kelompok III diberi perlakuan bonding mengandung klorheksidin, pada tiap kelompok dibagi menjadi 2 sub-kelompok yaitu kelompok tanpa direndam dan kelompok yang direndam NaOCl 10% selama satu jam, sehingga didapat enam sub-kelompok. Kemudian seluruh kelompok di ukur kekuatan ikat gesernya menggunakan Universal Testing Machine. Satu sampel dari setiap sub-kelompok dilakukan Scanning Electron Microscope (SEM). Data dianalisa statistik dengan uji hipotesis Kruskal Wallis yang dilanjutkan dengan uji Mann-Whitney. Hasil: rerata kekuatan ikat geser sebelum perendaman NaOCl 10% tertinggi kelompok I sedangkan rerata kekuatan ikat geser setelah perendaman NaOCl 10% tertinggi pada kelompok III. Terdapat perbedaan bermakna antara kelompok I terhadap kelompok II dan antara kelompok II terhadap kelompok III. Kesimpulan: Klorheksidin mempunyai efek terhadap pengurangan degradasi kekuatan ikat geser resin komposit-dentin.

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Resilience of composite resin-dentin bonding known as one of success composite resin restoration determinants. The purpose of this study was to analyze the effect of chlorhexidine on reducing the degradation of composite resin-dentin shear bond strength.

Methods: Twenty-four premolar crowns were divided into three groups then given different treatments. Group I was treated material bonding without chlorhexidine, group II was treated with chlorhexidine and bonding, group III was treated with chlorhexidine-contained bonding. Each group was divided into two sub-groups: the group without immersion of NaOCl 10% and the group with immersion of NaOCl 10% for one hour, then it were obtained six sub-groups. After twenty-four hours, shear bond strengths measured using Universal Testing Machine. A sample of each group was photographed with Scanning Electron Microscope (SEM). Statistical analysis was done by Kruskal Wallis test, then followed by Mann Whitney test to determine significance between groups.

Results: The mean value of shear bond strength before immersion of NaOCl 10% was highest on Group I, while the mean value of shear bond strength after immersion of NaOCl 10% was highest on Group III. There are significant differences between Group I with Group II and between Group II with Group III.

Conclusion: Chlorhexidine have an effect on reducing the degradation of shear bond strength of resin-dentin bonding."

"ABSTRAKLatar belakang: Ketahanan ikatan resin komposit-dentin merupakan salah satupenentu keberhasilan restorasi resin komposit. Tujuan penelitian ini adalahmenganalisis efek klorheksidin terhadap degradasi kekuatan ikat resin kompositdentin.Metode: Dua puluh empat sampel dentin yang diambil dari mahkota gigipremolar, dibagi menjadi tiga kelompok yang diberikan perlakuan berbeda.Kelompok I diberi perlakuan bahan bonding tanpa klorheksidin, kelompok IIdiberi perlakuan klorheksidin dan bonding, kelompok III diberi perlakuan bondingmengandung klorheksidin, pada tiap kelompok dibagi menjadi 2 sub-kelompokyaitu kelompok tanpa direndam dan kelompok yang direndam NaOCl 10%selama satu jam, sehingga didapat enam sub-kelompok. Kemudian seluruhkelompok di ukur kekuatan ikat gesernya menggunakan Universal TestingMachine. Satu sampel dari setiap sub-kelompok dilakukan Scanning ElectronMicroscope (SEM). Data dianalisa statistik dengan uji hipotesis Kruskal Wallisyang dilanjutkan dengan uji Mann-Whitney. Hasil: rerata kekuatan ikat gesersebelum perendaman NaOCl 10% tertinggi kelompok I sedangkan rerata kekuatanikat geser setelah perendaman NaOCl 10% tertinggi pada kelompok III. Terdapatperbedaan bermakna antara kelompok I terhadap kelompok II dan antarakelompok II terhadap kelompok III. Kesimpulan: Klorheksidin mempunyai efekterhadap pengurangan degradasi kekuatan ikat geser resin komposit-dentin.

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ABSTRACTBackground: Resilience of composite resin-dentin bonding known as one ofsuccess composite resin restoration determinants. The purpose of this study was toanalyze the effect of chlorhexidine on reducing the degradation of compositeresin-dentin shear bond strength. Methods: Twenty-four premolar crowns weredivided into three groups then given different treatments. Group I was treatedmaterial bonding without chlorhexidine, group II was treated with chlorhexidineand bonding, group III was treated with chlorhexidine-contained bonding. Eachgroup was divided into two sub-groups: the group without immersion of NaOCl10% and the group with immersion of NaOCl 10% for one hour, then it wereobtained six sub-groups. After twenty-four hours, shear bond strengths measuredusing Universal Testing Machine. A sample of each group was photographed withScanning Electron Microscope (SEM). Statistical analysis was done by KruskalWallis test, then followed by Mann Whitney test to determine significancebetween groups. Results: The mean value of shear bond strength beforeimmersion of NaOCl 10% was highest on Group I, while the mean value of shearbond strength after immersion of NaOCl 10% was highest on Group III. There aresignificant differences between Group I with Group II and between Group II withGroup III. Conclusion: Chlorhexidine have an effect on reducing the degradationof shear bond strength of resin-dentin bonding"